Dog diabetic food
Diabetic dog food
The relationship between obesity and NIDDM in humans is well documentated. Studies with dogs and cats have shown that a similar relationship exists in these species. Baseline plasma insulin level and insulin response to a glucose load increase linearly in dogs as a function of their degree of obesity. This effect occurs in both healthy and diabetic dogs and cats. Similarly, a study with cats found that healthy but obese cats had normal fasting plasma glucose concentrations, but the study showed abnormal results on glucose tolerance tests and slightly elevated baseline serum insulin concentrations. Significant delays in initial insulin response and substantially increased insulin responses at a later phase of the glucose tolerance test were found in the overweight cats.
Decreased tissue sensitivity to insulin and impaired beta-cell responsiveness to stimuli are believed to be the cause of these changes. Specifically, the tissue of obese animals has decreased numbers of cellular insulin receptors, and the receptors that are present have reduced binding affinity. In some cases, a post-receptor, intracellular defect in insulin action also occurs. Ultimately, these changes decrease the body's baseline insulin and insulin secretion increase in an attempt to compensate for the obesity-induced cellular resistance to insulin.
Weight reduction and control is an important aspect of the dietary management of diabetic animals that are overweight. When obesity is reduced in dogs and cats with abnormal insulin-secretory responses, glucose tolerance often improves. In addition, weight loss in pets with IDDM can result in enhanced tissue sensitivity to insulin, resulting in lowered daily insulin requirements. When a diabetic pet is overweight, caloric intake should be designed for weight loss and the eventual maintenance of ideal body weight. A diet that contains a high proportion of complex carbohydrates and reduced fat provides decreased energy density. However, a diet that is low in energy density must also contain adequate levels of all nutrients in forms that are available for digestion and absorption.
A commercial diet that is formulated to be complete and balanced while containing moderate fiber levels, increased complex carbohydrates, and reduced fat is recommended. Adding complex carbohydrates or fiber to a normal diet in a attempt to decrease energy density is contraindicated because this practice may cause increased stool volume, loose stools, or diarrhea and can lead to nutrient imbalances. When weight loss is instituted with pets that have IDDM, the pet's blood glucose should be carefully monitored and adjustments in insulin made as glucose tolerance improves.
We would love to hear your pet's story. Please add a comment.
The relationship between obesity and NIDDM in humans is well documentated. Studies with dogs and cats have shown that a similar relationship exists in these species. Baseline plasma insulin level and insulin response to a glucose load increase linearly in dogs as a function of their degree of obesity. This effect occurs in both healthy and diabetic dogs and cats. Similarly, a study with cats found that healthy but obese cats had normal fasting plasma glucose concentrations, but the study showed abnormal results on glucose tolerance tests and slightly elevated baseline serum insulin concentrations. Significant delays in initial insulin response and substantially increased insulin responses at a later phase of the glucose tolerance test were found in the overweight cats.
Decreased tissue sensitivity to insulin and impaired beta-cell responsiveness to stimuli are believed to be the cause of these changes. Specifically, the tissue of obese animals has decreased numbers of cellular insulin receptors, and the receptors that are present have reduced binding affinity. In some cases, a post-receptor, intracellular defect in insulin action also occurs. Ultimately, these changes decrease the body's baseline insulin and insulin secretion increase in an attempt to compensate for the obesity-induced cellular resistance to insulin.
Weight reduction and control is an important aspect of the dietary management of diabetic animals that are overweight. When obesity is reduced in dogs and cats with abnormal insulin-secretory responses, glucose tolerance often improves. In addition, weight loss in pets with IDDM can result in enhanced tissue sensitivity to insulin, resulting in lowered daily insulin requirements. When a diabetic pet is overweight, caloric intake should be designed for weight loss and the eventual maintenance of ideal body weight. A diet that contains a high proportion of complex carbohydrates and reduced fat provides decreased energy density. However, a diet that is low in energy density must also contain adequate levels of all nutrients in forms that are available for digestion and absorption.
A commercial diet that is formulated to be complete and balanced while containing moderate fiber levels, increased complex carbohydrates, and reduced fat is recommended. Adding complex carbohydrates or fiber to a normal diet in a attempt to decrease energy density is contraindicated because this practice may cause increased stool volume, loose stools, or diarrhea and can lead to nutrient imbalances. When weight loss is instituted with pets that have IDDM, the pet's blood glucose should be carefully monitored and adjustments in insulin made as glucose tolerance improves.
We would love to hear your pet's story. Please add a comment.
